Media Report
April 1, 2012Wayne State researcher seeks to link genome instability, chronic fatigue
http://detroit.cbslocal.com/2012/04/01/wayne-state-researcher-seeks-to-link-genome-instability-chronic-fatigue/
A national foundation has sought out a Wayne State University researcher in an effort to discover whether overall genome instability rather than a specific molecular mechanism may cause chronic fatigue and immune dysfunction syndrome (CFIDS). Henry Heng, associate professor at the Center for Molecular Medicine and Genetics and the Department of Pathology at WSU’s School of Medicine, recently received a $133,000 grant from the National CFIDS Foundation in collaboration with the Nancy Taylor Foundation for Chronic Diseases. Heng is to assess disease damage in patients with CFIDS, which also is known as chronic fatigue syndrome/myalgic encephalopathy (CFS/ME). He will use an advanced type of genomic testing to look for chromosomal aberrations or genomic instability in CFS/ ME patients. Heng believes scientists who have looked to single genes and mutations as the cause of diseases with complex sets of symptoms need to broaden their search to genomes.
A national foundation has sought out a Wayne State University researcher in an effort to discover whether overall genome instability rather than a specific molecular mechanism may cause chronic fatigue and immune dysfunction syndrome (CFIDS). Henry Heng, associate professor at the Center for Molecular Medicine and Genetics and the Department of Pathology at WSU’s School of Medicine, recently received a $133,000 grant from the National CFIDS Foundation in collaboration with the Nancy Taylor Foundation for Chronic Diseases. Heng is to assess disease damage in patients with CFIDS, which also is known as chronic fatigue syndrome/myalgic encephalopathy (CFS/ME). He will use an advanced type of genomic testing to look for chromosomal aberrations or genomic instability in CFS/ ME patients. Heng believes scientists who have looked to single genes and mutations as the cause of diseases with complex sets of symptoms need to broaden their search to genomes.
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